The GluN1, GluN2A, and GluN2B pre-M1 linker is intolerant to genetic
Por um escritor misterioso
Descrição
Common synaptic phenotypes arising from diverse mutations in the
GluN2A and GluN2B NMDA receptors use distinct allosteric routes
Positive allosteric modulators that target NMDA receptors rectify
Hodgkin–Huxley–Katz Prize Lecture: Genetic and pharmacological
Cross-subunit interactions that stabilize open states mediate
From bedside‐to‐bench: What disease‐associated variants are
Structure, Function, and Pharmacology of Glutamate Receptor Ion
Mechanistic Insight into NMDA Receptor Dysregulation by Rare
Molecular Mechanism of Disease-Associated Mutations in the Pre-M1
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